Vitamin D Joints

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Arthritis And Vitamin D: What The Research Says

Research has found that vitamin D may play a significant role in joint health, and that low levels may increase the risk of rheumatologic conditions such as arthritis. Several studies have found low blood levels of vitamin D in patients with osteoarthritis of the hip and knee. In another study of more than 2,000 people, researchers found that vitamin D deficiency was strongly associated with disabling symptoms among those with rheumatoid arthritis.

Why Do We Need Vitamin D?

This vitamin is crucial for bone health. A deficiency of vitamin D causes bones to soften and become weak. This is called osteomalacia in adults and rickets in children.
In addition, the vitamin may affect the immune system and could help to manage autoimmune conditions such as RA, Type 1 diabetes, and multiple sclerosis (MS).

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Vitamin D And Your Health

Not only does your body make vitamin D when your skin is exposed to the sun, but we also know vitamin D can ward off many health problems. Vitamin D is a naturally occurring compound that regulates the body’s use of calcium and phosphorus.
It’s crucial for the formation of bone and teeth. However, more information is needed to determine whether vitamin D supplements can help everyone living with chronic pain. Another study predicted that adults with a vitamin D deficiency who are older than 50 are more likely to develop pain in their hip and knee joints.
The study also noted that the pain is more likely to get worse if the deficiency isn’t treated. Other studies have concluded that people with RA have low vitamin D levels from their corticosteroid medications.

1. Introduction

Commonly affecting weight bearing synovial joints, OA is characterised by the degradation and loss of articular cartilage, abnormal subchondral bone growth and remodeling, and, in early stages, inflammation of the synovium.
There are, however, a range of risk factors known to associate with OA including age, gender, obesity, previous joint trauma, and genetics [1]. This is achieved by inducing the release of calcium into the circulation via increased bone turnover to prevent hypocalcaemia [5]. To date, numerous studies have shown its involvement and association with many aspects of the disease.

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