We will do our best to answer this and many other similar questions in this article which should ease your mind regarding this subject.
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Vitamin D toxicity is usually caused by large doses of vitamin D supplements — not by diet or sun exposure. That’s because your body regulates the amount of vitamin D produced by sun exposure, and even fortified foods don’t contain large amounts of vitamin D.
The main consequence of vitamin D toxicity is a buildup of calcium in your blood (hypercalcemia), which can cause nausea and vomiting, weakness, and frequent urination. Your doctor might also prescribe intravenous fluids and medications, such as corticosteroids or bisphosphonates.
This level is many times higher than the U.S. Recommended Dietary Allowance (RDA) for most adults of 600 IU of vitamin D a day. Review/update the information highlighted below and resubmit the form.
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1. Elevated Blood Levels
For example, in a 2020 case report, a 73-year-old man developed vitamin D toxicity after taking 10,000 IU of vitamin D per day for many years Note that 130,000 IU is over 30 times the generally recommended safe upper limit of 4,000 IU per day. Keep in mind that people who are low or deficient in vitamin D typically need to take much higher levels than the current Tolerable Upper Intake Level (UL) of 4,000 IU per day to reach and maintain optimal vitamin D levels. Toxicity symptoms have been reported at extremely high blood levels in cases where people took megadoses (very high doses) of vitamin D supplements for extended periods of time.
Symptoms
vitamin D can cause abnormally high levels of calcium in the blood. It occurs when you take in too much vitamin D. It’s usually the result of taking high-dose vitamin D supplements. And there has been an increase in overall hypervitaminosis D cases in the past few years.
You may not need to take additional calcium and vitamin D if you’re getting enough vitamin D from your multivitamin.
Msd Manual
a prohormone with several active metabolites that act as hormones. Vitamin D is metabolized by the liver to 25(OH)D (calcifediol, calcidiol, 25-hydroxycholecalciferol, or 25-hydroxyvitamin D), which is then converted by the kidneys to 1,25-dihydroxyvitamin D (1,25-dihydroxycholecalciferol, calcitriol, or active vitamin D hormone).
25(OH)D, the major circulating form, has some metabolic activity, but 1,25-dihydroxyvitamin D is the most metabolically active. The conversion to 1,25-dihydroxyvitamin D is regulated by its own concentration, parathyroid hormone (PTH), and serum concentrations of calcium and phosphat.